Using Marijuana May Not Raise the Risk of Using Harder Drugs
Marijuana is widely regarded as a "gateway" drug, that is, one whose
use results in an increased likelihood of using more serious drugs such
as cocaine and heroin. This gateway effect is one of the principal
reasons cited in defense of laws prohibiting the use or possession of
marijuana. A recent analysis by RAND's Drug Policy Research Center
(DPRC) suggests that data typically used to support a marijuana gateway
effect can be explained as well by a different theory. The new
research, by Andrew Morral, associate director of RAND Public Safety
and Justice, Daniel McCaffrey, and Susan Paddock, has implications for
U.S. marijuana policy. However, decisions about relaxing U.S. marijuana
laws must necessarily take into account many other factors in addition
to whether or not marijuana is a gateway drug.
Support for the Gateway Effect
Although marijuana has never been shown to have a gateway
effect, three drug initiation facts support the notion that marijuana
use raises the risk of hard-drug use:
- Marijuana users are many times more likely than nonusers to progress to hard-drug use.
- Almost all who have used both marijuana and hard drugs used marijuana first.
- The greater the frequency of marijuana use, the greater the likelihood of using hard drugs later.
This evidence would appear to make a strong case for a
gateway effect. However, another explanation has been suggested: Those
who use drugs may have an underlying propensity to do so that is not
specific to any one drug. There is some support for such a
"common-factor" model in studies of genetic, familial, and
environmental factors influencing drug use. The presence of a common
propensity could explain why people who use one drug are so much more
likely to use another than are people who do not use the first drug. It
has also been suggested that marijuana use precedes hard-drug use
simply because opportunities to use marijuana come earlier in life than
opportunities to use hard drugs. The DPRC analysis offers the first
quantitative evidence that these observations can, without resort to a
gateway effect, explain the strong observed associations between
marijuana and hard-drug initiation.
New Support for Other Explanations
The DPRC research team examined the drug use patterns reported
by more than 58,000 U.S. residents between the ages of 12 and 25 who
participated in the National Household Surveys on Drug Abuse (NHSDA)
conducted between 1982 and 1994.[1]
Using a statistical model, the researchers tested whether the observed
patterns of drug use initiation might be expected if drug initiation
risks were determined exclusively by
- when youths had a first opportunity to use each drug
- individuals' drug use propensity, which was assumed to be normally distributed[2] in the population
- chance (or random) factors.
To put it another way, the researchers addressed the question: Could
the drug initiation facts listed in the first section of this brief be
explained without recourse to a marijuana gateway effect?
Figure 1—Probabilities of Initiating Hard Drugs, Marijuana Users and Nonusers |
The research team found that these associations could be explained without any gateway effects:
- The statistical model could explain the increased risk of
hard-drug initiation experienced by marijuana users. Indeed, the model
predicted that marijuana users would be at even greater risk of drug
use progression than the actual NHSDA data show (see Figure 1).
- The model predicted that only a fraction of hard-drug
users would not have tried marijuana first. Whereas in the NHSDA data
1.6 percent of adolescents tried hard drugs before marijuana, the model
predicted an even stronger sequencing of initiation, with just 1.1
percent trying hard drugs first.
- The modeled relationship between marijuana use frequency
and hard-drug initiation could closely match the actual relationship
(see Figure 2).
The new DPRC research thus demonstrates that the phenomena supporting
claims that marijuana is a gateway drug also support the alternative
explanation: that it is not marijuana use but individuals'
opportunities and unique propensities to use drugs that determine their
risk of initiating hard drugs. The research does not disprove the
gateway theory; it merely shows that another explanation is plausible.
Figure 2—Probabilities of Hard-Drug Initiation, Given Marijuana Use Frequency in the Preceding Year |
Some might argue that as long as the gateway theory remains a possible
explanation, policymakers should play it safe and retain current
strictures against marijuana use and possession. That attitude might be
a sound one if current marijuana policies were free of costs and harms.
But prohibition policies are not cost-free, and their harms are
significant: The more than 700,000 marijuana arrests per year in the
United States burden individuals, families, neighborhoods, and society
as a whole.
Marijuana policies should weigh these harms of prohibition against the
harms of increased marijuana availability and use, harms that could
include adverse effects on the health, development, education, and
cognitive functioning of marijuana users. However, the harms of
marijuana use can no longer be viewed as necessarily including an
expansion of hard-drug use and its associated harms. This shift in
perspective ought to change the overall balance between the harms and
benefits of different marijuana policies. Whether it is sufficient to
change it decisively is something that the new DPRC research cannot aid
in resolving.
[1]In subsequent years, respondents have not been asked about their first opportunity to use various drugs.
[2]That is, some people have a high or low propensity, but most people have a propensity near the middle of the range.
RB-6010 (2002)RAND research briefs summarize research
that has been more fully documented elsewhere. This research brief
describes work done in RAND's Drug Policy Research Center, a joint endeavor of RAND Public Safety and Justice and RAND Health.
The research is documented in "Reassessing the Marijuana Gateway
Effect" by Andrew R. Morral, Daniel F. McCaffrey, and Susan M. Paddock,
Addiction 97:1493-1504, 2002.
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